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Another adverse effect of cancer chemotherapy is known as tumour lysis syndrome. This is a collection of symptoms caused by the death of many cells in the tumour. A significant symptom is the production of large amounts of uric acid as a result of the breakdown of nucleic acids. High plasma levels of uric acid can precipitate gout, or interfere with treatment for pre-existing gout, or uric acid can deposit in the kidneys and cause kidney damage.

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Common adverse effects include dizziness, drowsiness, headache, rash, bruising, and gastrointestinal upset. Archived from the original on 22 July Annals of the Rheumatic Diseases. It is not to be confused with Analgin. Otherwise it is Schedule 4 Prescription only. Robin Analogue-based Drug Discovery. In other projects Wikimedia Commons.

OTC preparations of naproxen in the U. Naproxen may have cheap celebrex canada activity against influenza. Process Res. Archived from the original PDF on 21 September Bayer Health Care. November The Journal of Biological Chemistry. Drug Metabolism and Disposition. London, UK: Pharmaceutical Press. Naproxen was patented in, and approved for medical use in the United States in Naproxen sodium is used as a bridge therapy in medication-overuse headache to slowly take patients off other medications.

Aleve, Naprosyn, others. Analgesics N02A, N02B. Pyrazolidines Clofezone Mofebutazone Oxyphenbutazone Phenylbutazone. Australian Medicines Handbook ed. Naproxen, ibuprofen, and benoxaprofen. Retrieved 11 March British Medical Association. Clinical Correlations. Interactive image. Conversely, naproxen was associated with higher rates of upper gastrointestinal bleeding complications compared with other NSAIDs.

January August Journal of Internal Medicine. Syntex first marketed naproxen in, as the prescription drug Naprosyn.

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Patients with unipolar depression are effectively treated with drugs, such as chlorimipramine and more recently fluoxetine, which are known to block neuronal uptake of serotonin. In addition, giving alone or in combination with chlorimipramine seems to benefit these patients. Evidence against this serotonin theory includes the fact that low levels of ( have recovered and in some patients with mania. It seems likely that low levels of serotonin or noradrenaline do not cause depression, but that more complicated mechanisms involving a change in balance between these monoamines are involved. Despite evidence for and against, there remains one major problem with the monoamine theory of depression. All antidepressants take weeks to have an effect, which is far longer than it takes to alter brain amines.

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Amantadine has few side effects, mainly gastrointestinal disturbances and ankle oedema, but becomes less effective over time as the dopaminergic neurones are progressively lost. Monoamine oxidase is an enzyme that metabolizes monoamines.

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Cholinergic systems may also be involved in depression since many antidepressants have strong anti-cholinergic activity. High doses of adrenocorticotrophic hormone or cortisol can produce depression and depressed patients frequently have raised levels of cortisol in plasma.

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The effect of this is to combine a reduction in heart rate with a fall in blood pressure and they are useful in treating angina and hypertension.

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