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What is Xanax? How does it work mechanism of action? Alprazolam Xanax XR, Niravam, is an anti- anxiety medication in the benzodiazepine drug family, the same family that includes diazepam Valium, clonazepam Klonopin, lorazepam Ativan, flurazepam Dalmane, and others. Alprazolam and other benzodiazepines act by enhancing the effects of gamma-aminobutyric acid GABA in the brain. GABA is a neurotransmitter a chemical that nerve cells use to communicate with each other that inhibits activity in the brain.

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Alprazolam is a depressant whose effects include anxiety suppression, sedation, disinhibition, and muscle relaxation. Alprazolam has a fast onset of action and symptomatic relief. Ninety percent of peak effects are achieved within the first hour of using in preparation for panic disorder and full peak effects are achieved in 1. Alprazolam is capable of producing strong sedation and can lead to a lethargic state. This is usually preceded by loss of motor skills and coordination with the effect ranging from motor impairment and difficulty moving at lower doses to complete lethargy or inability to stand up or move at high doses.

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Sulphonamides interact with warfarin by enhancing the actions of these drugs. The most important adverse effects of sulphonamides are rashes, renal failure and various blood disorders. Trimethoprim inhibits another enzyme, dihydrofolate reductase, in the same folic acid metabolic pathway. Folic acid is converted into folate, which then has to be converted into an activated form by dihydrofolate reductase. In this way, trimethoprim interferes with the conversion of folate into its activated form, which is a cofactor in the synthesis of bacterial DNA.

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