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Your medication may look different. If you have questions, ask your pharmacist. Pronunciation FEN-ter-meen. Losing weight and keeping it off can lessen the many health risks that come with obesity, including heart disease, diabetes, high blood pressure, and a shorter life. It is not known how this medication helps people to lose weight.

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There may be involvement of defective nicotinic receptors because the use of transdermal nicotine patches has been shown to improve some symptoms of the disorder. Behavioural therapy or counselling may be helpful in some individuals with ADHD. Psychostimulant drugs are used to treat ADHD, where paradoxically they have a calming effect and improve concentration and attentiveness in about should be under the guidance of a specialist in ADHD. Examples of drugs used to treat ADHD are dexamfetamine, methylphenidate and atomoxetine. These drugs inhibit the re-uptake of noradrenaline and dopamine. Increase in levels of these two transmitters in the prefrontal cortex is thought to increase inhibitory control in the limbic system.

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These drugs are known as antisense oligonucleotides, which are small synthetic pieces of DNA that are complementary to a segment of mRNA. A hybrid of ON DNA and mRNA forms, which prevents translation of the mRNA. This means that the effect of the original oncogene is blocked. Cancer cells produce enzymes that can breakdown the extracellular matrix between normal cells and allow cancer cells to invade and spread in normal tissue. New drugs are being developed that inhibit these enzymes. In order for tumours to grow they need to develop a blood supply.

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Their use depends on their ability to inhibit the activity and proliferation of lymphocytes and other leukocytes and because of this they are very toxic to the bone marrow. Other adverse effects are nausea, leukopenia, blurred vision, rashes and hair loss. Cytokines are chemicals released by damaged cells and cells involved in inflammation and repair. Drugs have been developed against two of them, tumour necrosis factor α (TNF-α) and interleukin and together they are key players in the inflammatory response. Levels of both are raised in rheumatoid arthritis and other autoimmune diseases. Cytokine inhibitors are relatively new and they must only be prescribed under specialist supervision.

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(Note: a nucleoside is a base [either purine or pyrimidine] plus a sugar molecule; a nucleotide is a base plus a phosphorylated sugar molecule. See Figure for a diagram of a nucleotide.

She is aware that she suffers from a disorder with a long name, which makes her muscles weak.
Loss or mutation of tumour suppressor genes allows cells to undergo malignant transformations.
This includes making more viral RNA and viral proteins, which are assembled into new virus particles.
It is up to individuals to negotiate with their employer that supplementary prescribing should form part of their job.
This increases blood volume and restores blood pressure.
Fluorouracil is used to treat gastrointestinal and breast cancers, often together with synthetic folinates.
This results in a reduction in DNA synthesis and particularly affects rapidly dividing cells.

See Figure for a diagram of a nucleotide. ) Aciclovir is an analogue of guanosine nucleoside.

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Chemically, most drugs are either weak acids or weak bases. In an acidic environment, as in the stomach, acidic drugs are unionized according to the following simple equation: where A is an acidic drug and the excess hydrogen ions drive the equation to the left.

Treatment of osteomalacia and rickets is by vitamin D supplements. Some drugs, for example anticonvulsants phenytoin, phenobarbital and corticosteroids can lead to osteomalacia and rickets by depressing vitamin D dependent calcium uptake in the intestine. Myasthenia gravis is an autoimmune disorder, which is diagnosed in about people every year in the United Kingdom, mainly affecting women between Normal Myasthenia gravis nerve terminals muscle cell membrane mitochondrion junctional folds vesicles containing acetylcholine acetylcholine receptors acetylcholinesterase skeletal muscle is disrupted. Normally when impulses travel down a neuron to skeletal muscle the nerve endings release acetylcholine, which crosses the neuromuscular junction and brings about the activation of muscle contraction. In myasthenia gravis the receptors for acetylcholine on the muscle cell membrane are progressively destroyed by antibodies. The antibodies are produced by the patients own immune system resulting in an autoimmune reaction, which causes structural changes at the synapse.

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Treatment aims to enhance cholinergic transmission. The most useful drugs are central acetylcholinesterase inhibitors, for example donepezil. Acetylcholinesterase is the enzyme that normally breaks down acetylcholine after it has interacted with its receptors at the synapse. Inhibition of this enzyme in the brain increases the amount of acetylcholine available and prolongs its action. These drugs produce a modest improvement in memory or slow progression of symptoms in some patients. The response to anti-cholinesterase drugs may take several weeks.