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A new study in the October 18, issue of the venerable New England Journal of Medicine assesses the efficacy of drugging dementia patients with Risperdal versus abruptly discontinuing the drug and substituting a placebo. Despite confirming the high risk of harm and death when giving Risperdal to people with dementia, the authors primarily focused on the second group of patients — those who were started on Risperdal and abruptly stopped. The authors indicate this risk of relapse should factor against having Risperdal withdrawn from patients with dementia. Risperdal, and other antipsychotics are well known and increasingly notorious for rendering many patients with dementia into a near-vegetative state. When a patient is sedated and subdued, he or she will of course appear to be less agitated or psychotic. When the drug is suddenly stopped, the brain may be left risperdal mail order excess dopamine, which can itself induce temporary psychosis. Beyond the neurophysiologic impact of abrupt Risperdal cessation, taking a person with dementia off of a sedating drug, without doing anything more, can be expected to potentially lead to incidents of agitation and aggression.
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This still does not explain the swings in mood and little is known about how or why alternation between depression and mania occurs. Patients with unipolar depression are effectively treated with drugs, such as chlorimipramine and more recently fluoxetine, which are known to block neuronal uptake of serotonin.
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Apart from the possibility of adverse drug reactions occurring, individuals show variation in response to drugs and this is particularly likely in the very young and the very old. In order to have an effect on the body, therapeutic, adverse or otherwise, a drug must interact with its site of action.
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