Your doctor may prescribe Crestor to lower your risk of stroke, heart attack, or other cardiac complications, which can occur in those who have diabetes, coronary heart disease, or other risk factors. Using Crestor alone is not enough: You will also have to eat a healthy diet and make other lifestyle changes like exercising, quitting smoking, and maintaining a healthy weight. Even if you make all of these lifestyle changes, it may still take up to four weeks before you get the full benefit of Crestor. People of Asian descent should be aware that they might absorb this drug at a faster rate than other people. As a result, your doctor might need to put you on a lower-than-normal starting dose. In some rare cases, people using Crestor have developed a disorder in which muscle tissue breaks down, resulting in kidney failure. This is more likely to happen if you are older or if you have kidney disease or an underactive thyroid hypothyroidism that is poorly controlled.

Metrics details. Many patients at high risk of cardiovascular disease do not achieve recommended low-density lipoprotein cholesterol LDL-C goals. This study compared the efficacy and safety of low doses of rosuvastatin 10 mg and atorvastatin 20 mg in high-risk patients with hypercholesterolemia. The primary endpoint was the percentage change from baseline in LDL-C levels at 6 weeks. Rosuvastatin 10 mg reduced LDL-C levels significantly more than atorvastatin 20 mg at week 6 High-density lipoprotein cholesterol was increased significantly with rosuvastatin 10 mg versus atorvastatin 20 mg 6.

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Rosuvastatin calcium is a white amorphous powder that is sparingly soluble in water and methanol, and slightly soluble in ethanol. Lipid-altering agents should be used in addition to a diet restricted in saturated fat and cholesterol when response to diet and nonpharmacological interventions alone has been inadequate. The usual starting dose is 10 to 20 mg once daily. The usual starting dose in adult patients with homozygous familial hypercholesterolemia is 20 mg once daily. The tablet should be swallowed whole. After initiation or upon titration of CRESTOR, lipid levels should be analyzed within 2 to 4 weeks and the dosage adjusted accordingly. In heterozygous familial hypercholesterolemia, the recommended dose range is 5 to 10 mg orally once daily in patients 8 to less than 10 years of age, and 5 to 20 mg orally once daily in patients 10 to 17 years of age.

Although the recommending starting Crestor dosage is 10 mg once a day, your healthcare provider may recommend a lower Crestor dose 5 mg per day if you meet certain criteria Adults—5 to 40 milligrams mg once a day. These medications are not usually taken together. Rosuvastatin marketed as Crestor 10 mg tablets. They may be naturally resistant to Crestor side effects. Children 8 to 9 years of age—5 to 10 mg per day Crestor rosuvastatin crestor 20 mg range from strengths of 5 mg to 40 mg. Crestor Side Effects: Headache.

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Back to Medicines A to Z. Your doctor may also prescribe rosuvastatin if you have diabetes, kidney disease, or a family history of rheumatoid arthritis or heart disease. Rosuvastatin also comes as a higher 40mg dose for adults. It's not suitable for some people. Tell your doctor if you also.

We will ship your order as soon as international postal services resume. Orders are expected to start shipping on April 23rd. If you have urgent needs, please obtain from your local pharmacy. Keep me up to date with shipping announcements. Crestor contains rosuvastatin, a cholesterol-lowering rosuvastatin crestor 20 mg that is used to treat hypercholesterolaemia by lowering high levels of cholesterol and triglycerides in the blood, which helps reduce risk of heart disease. Crestor tablets 20mg are used to lower high levels of cholesterol hypercholesterolaemia and other fats triglycerides in the blood to reduce health risks associated with coronary heart disease CHD, including heart attack and angina that can be caused by high cholesterol.

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What is rosuvastatin, and how does it work mechanism of action? Rosuvastatin is an oral drug for lowering blood cholesterol levels. These drugs reduce cholesterol levels by inhibiting HMG-CoA reductase, an enzyme that produces cholesterol in the liver. Rosuvastatin and other statins lower blood total cholesterol as well as blood LDL cholesterol levels. LDL cholesterol is the bad type of cholesterol that increases the risk of coronary artery disease atherosclerosis and heart attacks.

Each tablet contains Round, yellow coloured tablets, intagliated with 'ZD' and '5' on one side and plain on the reverse. Round, pink coloured tablets, intagliated with 'ZD' and '10' on one side and plain on the reverse. Round, pink coloured tablets, intagliated with 'ZD' and '20' on one side and plain on the reverse. Oval, pink coloured tablets, intagliated with 'ZD' on one side and '40' on the reverse. Adults, adolescents and children aged 6 years rosuvastatin crestor 20 mg older with primary hypercholesterolaemia type IIa including heterozygous familial hypercholesterolaemia or mixed dyslipidaemia type IIb as an adjunct to diet when response to diet and other non-pharmacological treatments e.

After rosuvastatin crestor 20 mg or upon titration, analyze lipid levels within weeks and adjust dosage accordingly. Indicated as adjunctive therapy to diet to slow the progression of atherosclerosis in adult patients as part of a treatment strategy to lower total cholesterol and LDL cholesterol to target levels. Increased blood glucose and glycosylated hemoglobin HbA1c levels reported with statin intake; in some instances, these increases may exceed the threshold for the diagnosis of diabetes mellitus. Discontinue treatment if markedly elevated creatine kinase levels occur or myopathy is diagnosed or suspected; temporarily withhold in any patient with an acute, serious condition suggestive of myopathy or predisposing to the development of renal failure secondary to rhabdomyolysis eg, sepsis; hypotension; dehydration; major surgery; trauma; severe metabolic, endocrine, or electrolyte disorders; uncontrolled seizures. Rare reports of immune-mediated necrotizing myopathy, characterized by increased serum creatine kinase that persists despite discontinuing statin.

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