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It is used to treat Type 2 diabetes, heart disorders.
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It helps control blood sugar levels. Anorexia, nausea, vomiting, diarrhoea, wt loss, flatulence, occasional metallic taste; weakness; hypoglycaemia; rash, malabsorption of vit B Chest discomfort, flushing, palpitation, chills, headache, lightheadedness, indigestion, abdominal discomfort. Potentially Fatal: Lactic acidosis in presence of renal failure and alcoholism. Additive effect with sulphonylureas. Glycaemic control may be affected by diuretics, corticosteroids, phenothiazines, thyroid products, oestrogens, oral contraceptives, phenytoin, nicotinic acid, sympathomimetics, calcium channel blockers, chlorpromazine and isoniazid. May decrease the anticoagulant effect of phenprocoumon, therefore routine anticoagulant monitoring is recommended. Potentially Fatal: Lactic acidosis with alcohol. Acute or chronic metabolic acidosis with or without coma including diabetic ketoacidosis.
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It works by lowering glucose production in the liver, delaying the absorption of sugar glucose from the intestines and increasing the body's sensitivity to insulin. Anuj Saini. Varun Gupta. Gluconorm Lupin Ltd. Cetapin Sanofi India Ltd. Melmet Micro Labs Ltd.
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The advantage of this is that a patient will need to take pills less often. It also means there may be fewer side-effects as the levels of the of drug in the body are more consistent. NDMA is classified as a probable human carcinogen — a substance that could cause cancer — based on results from laboratory tests. NDMA is a known environmental contaminant and in lesser quantities, it is found in water and foods, including meats, dairy products and vegetables. India, known as the diabetic capital of the world, has around 7.
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It works by decreasing glucose production by the liver and increasing the insulin sensitivity of body tissues. French physician Jean Sterne began study in humans in the s. It was introduced as a medication in France in and the United States in It is also used in the treatment of polycystic ovary syndrome. It is not associated with weight gain.
This was probably because the majority of viral infections are eliminated by the hosts immune system. With the advent of the HIV and acquired immunodeficiency syndrome that follows infection with HIV, many new antiviral drugs have been introduced since the end of the about how viruses replicate inside cells and how they affect cell function, then no doubt more antiviral drugs will be developed. Antiviral drugs have been developed that work by the following mechanisms: inhibition of the synthesis of viral DNA, RNA and proteins (for example, nucleoside analogues, reverse transcriptase inhibitors, protease inhibitors); reduction in release of viral genetic material once inside the metformin buy india cell (for example, amantadine); interference with penetration of virus through the cell membrane (for example, neuraminidase inhibitors); interference with attachment of the virus to the host cell membrane (for example, vaccines, immunoglobulins and interferons). Resistance can occur through a number of consequences of viral mutation. There may be reduced phosphorylation of the drug, alteration of viral enzyme targets, or alteration of target channel protein.
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Viruses are intracellular parasites; they cannot replicate themselves without using the contents of the host cell to synthesize the cellular components necessary for their reproduction. This makes the development of effective antiviral drugs that do not damage healthy host cells very difficult.
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